The mechanism of action of Florest in obsessive compulsive disorder is presumed to be linked to its specific serotonin reuptake inhibition in brain neurons. Florest has been shown to be a potent inhibitor of the serotonin reuptake transporter in preclinical studies, both in-vitro and in-vivo. In in-vitro studies, Florest had no significant affinity for histaminergic, alpha or beta adrenergic, muscarinic, or dopaminergic receptors. Antagonism of some of these receptors is thought to be associated with various sedative, cardiovascular, anticholinergic, and extrapyramidal effects of some psychotropic drugs.